Most people would think that the cause of diabetes, heart disease, or other chronic diseases is due to their own unhealthy lifestyle decisions. For example, most people would think that smoking, stress, and lack of exercise would make them more likely to contract such diseases. However, Barker has a different point of view. As DJP Barker states in his article entitled, Fetal and Infant Origins of Adult Disease Hypothesis, that the adaptations that the fetus makes when undernourished rather than the adaptations made in adult life and those made during early development tend to have a permanent effect on the structure and function of the body. (Barker, 2001). He hypothesizes that low birth weight is a contributing factor to the development of coronary heart disease in adulthood, but overlooks that there are sociological factors that during adult life can also trigger the development of coronary heart disease. Although the fetal origins hypothesis uses a biological explanation for the development of coronary heart disease, it fails to recognize the sociological factors of coronary heart disease. One's position on the socioeconomic gradient contributes to the risk of coronary heart disease. According to Barker's hypothesis, he explains how the mother's exposure and metabolism can impact the health of the fetus. He hypothesizes that the environment the mother is exposed to before and during birth may have an effect on the health of the fetus as it ages. As we know, the female is responsible for reproduction, so the health of the mother can have a direct effect on the fetus. If the mother is undernourished, it will potentially send a signal to the fetus that the environment it is about to enter will be harsh and that the… center of the card… dual relationship with cardiovascular disease. Overall, the results show that it is better to have a growing or stable income to decrease the risk of cardiovascular disease. However, Johnson's hypothesis only used socioeconomic status in relation to income with coronary heart disease, but does not explain how coronary heart disease would be measured if you have no income, for example as children and adolescents, and therefore what the risk would be for coronary heart disease disease would be a measure. Would it then be a measure in relation to the parents' income? Since children are cared for by their parents, whatever the parent is exposed to will have an indirect relationship with the children. Follow-up studies should be conducted on how the social gradient changes across the lifespan in relation to cardiovascular disease.